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in which focal lymphocytic infiltration leads to damage of myelin and axons. ""The location close to Newport downtown, stores, restaurant. Because flexibility matters.In the larger Providence area, you'll find 6 inexpensive motels among more than 119 hotels and other accommodations. Additional terms may apply.By saving on your room price, you can enjoy more of what Cranston has to offer. Leyden Farm ..."Close to the highway, good price, clean, microwave and fridge. Fantasyland and Monster Mini Golf are also within 2 mi (3 ..."yhhyuijhgttggttttttttttttttttttttttttttttttttttttttttt""yhhyuijhgttggttttttttttttttttttttttttttttttttttttttttt"Situated in Wrentham, this motel is within 1 mi (2 km) of Plainridge Race Course and Plainridge Park Casino. Motel 6 Seekonk, MA - Providence East: Central Cranston is 8.4 mi (13.5 km) away. Favorite places in and around Cranston include Twin River Casino, Dunkin’ Donuts Center, and Providence Performing Arts Center.
Thames Street and Easton's ..."Very clean. Newport Mansions is ...Situated near the airport, this motel is 0.6 mi (1 km) from Newport Playhouse and within 3 mi (5 km) of Thames Street and Easton's Beach.
"Situated in Somerset, this motel is within 3 mi (5 km) of USS Massachusetts, Battleship Cove, and Narrows Center for the Arts.
Rodeway Inn Middletown: 20.3 mi (32.7 km) from the heart of Cranston. Department of Clinical Neurosciences, University of Cambridge Clinical School, Addenbrooke's Hospital, Cambridge, UKCorrespondence to: Dr Alasdair Coles, University of Cambridge Clinical School, Addenbrooke's Hospital, Hills Road, Cambridge CB2 2QQ, UKDepartment of Clinical Neurosciences, University of Cambridge Clinical School, Addenbrooke's Hospital, Cambridge, UK An association of gadolinium leakage with clinical and electrophysiological deficits.Long-term protection of central axons with phenytoin in monophasic and chronic-relapsing EAE.Axonal protection in multiple sclerosis—a particular need during remyelination?.The restoration of conduction by central demyelination.Familial effects on the clinical course of multiple sclerosis.The natural history of multiple sclerosis: a geographically based study 9: observations on the progressive phase of the disease.Nitric oxide donors reversibly block axonal conduction: demyelinated axons are especially susceptible.Electrically active axons degenerate when exposed to nitric oxide.Different pathways for iNOS-mediated toxicity in vitro dependent on neuronal maturation and NMDA receptor expression.Mitochondrial dysfunction as a cause of axonal degeneration in multiple sclerosis patients.Disruption of neurofascin localization reveals early changes preceding demyelination and remyelination in multiple sclerosis.A role for oligodendrocyte-derived IGF-1 in trophic support of cortical neurons.Oligodendrocytes promote neuronal survival and axonal length by distinct intracellular mechanisms: a novel role for oligodendrocyte-derived glial cell line-derived neurotrophic factor.Trophic factors attenuate nitric oxide mediated neuronal and axonal injury in vitro: roles and interactions of mitogen-activated protein kinase signalling pathways.Leber's hereditary optic neuropathy mitochondrial DNA mutations in multiple sclerosis.Occurrence of a multiple sclerosis-like illness in women who have a Leber's hereditary optic neuropathy mitochondrial DNA mutation.The clinical features of Leber's hereditary optic neuropathy defined by the presence of a pathogenic mitochondrial DNA mutation.Heterogeneity of multiple sclerosis lesions: implications for the pathogenesis of demyelination.Distinct patterns of multiple sclerosis pathology indicates heterogeneity on pathogenesis.Mitochondrial defects in acute multiple sclerosis lesions.Homogeneity of active demyelinating lesions in established multiple sclerosis.A role for humoral mechanisms in the pathogenesis of Devic's neuromyelitis optica.Tissue preconditioning may explain concentric lesions in Balo's type of multiple sclerosis.A serum autoantibody marker of neuromyelitis optica: distinction from multiple sclerosis.IgG marker of optic-spinal multiple sclerosis binds to the aquaporin-4 water channel.The clinical course of neuromyelitis optica (Devic's syndrome).Revised diagnostic criteria for neuromyelitis optica.Pattern-specific loss of aquaporin-4 immunoreactivity distinguishes neuromyelitis optica from multiple sclerosis.Loss of aquaporin 4 in lesions of neuromyelitis optica: distinction from multiple sclerosis.Relation between humoral pathological changes in multiple sclerosis and response to therapeutic plasma exchange.Heterogeneity of aquaporin-4 autoimmunity and spinal cord lesions in multiple sclerosis in Japanese.Changes in the clinical phenotypes of multiple sclerosis during the past 50 years in Japan.Role of return migration in the emergence of multiple sclerosis in the French West Indies.The marvellous harmony of the nervous parts: the origins of multiple sclerosis.A meta-analysis of methylprednisolone in recovery from multiple sclerosis exacerbations.A randomized trial of plasma exchange in acute central nervous system inflammatory demyelinating disease.Plasma exchange for severe attacks of CNS demyelination: predictors of response.Randomised double-blind placebo-controlled study of interferon beta-1a in relapsing/remitting multiple sclerosis.Intramuscular interferon beta-1a for disease progression in relapsing multiple sclerosis.Interferon beta-1b in the treatment of multiple sclerosis: final outcome of the randomized controlled trial.Copolymer 1 reduces relapse rate and improves disability in relapsing-remitting multiple sclerosis: results of a phase III multicenter, double-blind placebo-controlled trial.
clinical correlate of which is progressive accumulation of disability. All numbers are air distances – as the crow flies/great circle distance.
Clean, everything worked. International ...Situated in Middletown, this motel is 2.9 mi (4.7 km) from Naval War College Museum and within 6 mi (10 km) of Touro Synagogue and Easton's Beach. Traveled during COVID and they definitely seemed to be compliant to new standards.